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06/02/2026

Preeclampsia is not a single disease; it's a multi-system dysfunction.

Clinically, it’s defined as new‑onset hypertension after 20 weeks with proteinuria, and/or maternal organ dysfunction, and/or uteroplacental dysfunction such as fetal growth restriction. In classic, placenta‑driven preeclampsia, this pattern reflects injury at the maternal–fetal interface, whether the primary driver is maternal vascular disease, fetal/placental factors, or both.

Emerging research is helping us to recognize distinct preeclampsia patterns that point to whether the origin is more maternal or fetal, with the common thread being that preeclampsia is placental-driven.

However, some pregnancies look and behave like preeclampsia (Hypertension, proteinuria, abnormal labs, additional symptoms....) but are not placenta. These are preeclampsia mimickers...conditions that cause systemic maternal dysfunction or symptoms that are akin to preeclampsia but are not actually preeclampsia.

Over the next few weeks, I am going to cover what the research says about using standard labs to differentiate true preeclampsia from PE-mimickers and help you distinguish underlying etiology.

The intent is to move past basic symptom-checklist diagnosis and into true pattern recognition...using the labs you are already drawing and introducing you to some new ones.